Notice almost everyone who takes antidepressants. Is it the depression or the medication that is responsible? responsible? Experts are quick to agree that it is the disorder. But is that true?
What’s the truth?
Some people have less desire to sex while others are more receptive (are harder to get started), some are impotent and many feel so uncomfortable talking about the subject that they won’t even consider it. Others feel pain while having sex and many do not reach a peak – even when masturbating.
What can you do? Sexuality can be a delicate topic. It is a sensitive topic that few people seek help for, and very few people openly address it. It is common to think that “Maybe it’s just you” is the answer. This is a common thought. It is more common for women to assume that they can do it themselves and wonder if their love has changed. Men of these women often feel rejected and respond very differently.
It is more difficult for men who don’t feel the need to be happy. Some people see it as a way of liberating themselves. Those who feel no desire are particularly vulnerable. Others feel like complete failures. They could rely on their sexuality before. The suicide risk of men who leave for the first weekend after four weeks of being stationary is high, according to Wolfersdorf’s study. It is difficult to imagine what was attempted during that weekend, and how it failed.
Does a disturbed sexual life make it harder to get over depression? It is the opposite. Positive effects of healthy sexuality on depression symptoms. First, antidepressants can cause sexual dysfunction much more consistently than antidepressant effects. Professor Peter Gotzsche states that antidepressants trigger sexual dysfunction, which is why he calls them “highly effective means to disrupt the sexual life”.
Although there may be many causes for Impotence, Eroxel helps you improve your sex life by giving you a powerful Boost: Eroxel
Many people believe that the symptoms of antidepressants include a disturbance in sexual function. This is a false assumption. Two mechanisms are responsible for the disturbance of sexuality:
1. Drugs pass through the stomach to the small intestine. The active ingredient is then released and transported to other locations with the help the blood. Initially, however, there are a lot more active ingredients in the intestinal area, which leads to more serotonin being produced there. Serotonin does not come from the brain, but is mostly found in the gastrointestinal region. area. Here 95 percent of the body’s serotonin can be found. Most of the serotonin transporter proteins that are biochemical targets for drugs are also found in the intestinal area. The function of serotonin in the intestinal area is to contract smooth muscle. Serotonin’s effect on muscles does not have anything to do with depression. There is an SSRI that can be prescribed for antidepressant purposes, but it is only recommended by urologists. It’s called dapoxetine, and it is used to prevent premature ejaculation.
2. Too much serotonin can prevent sexuality. However, too little or too much serotonin can lead to hypersexuality or nymphomaniacs. Animal experiments using TPH2-knockout mice have shown this. Researchers made a surprising discovery in these animals. The TPH2-knockout animals performed well in standard depression tests, which pharmacologists use for determining the effectiveness of antidepressants. These animals are not depressed, even though they have no serotonin.
In principle, physicians – because the sexuality-restricting side effects have been known for a very long time – would have to take this frequently occurring side effect into account, especially if the patient is living in a partnership. Therefore, the doctor must choose an antidepressant that doesn’t trigger sexual dysfunction.
However, that is wishful thinking. It does not happen. Doctors will most often recommend SSRIs. Why is this? Because of an incorrect prescription in the U.S., and the resulting data that were adopted for Germany. The U.S. Food and Drug Administration requires that side effects be addressed by the patient. Only a little less than 10 percent is required for sexual dysfunction. This is why it is the official number. Sexual dysfunction is not considered to to be a common condition or to be very serious. These disorders are not common, but they do occur.
Yes, it is. You will receive different answers depending on who you ask. Montejo’s 1997 and 2001 studies on sexual dysfunction are the most well-known. Montejo stated that sexual dysfunction was more common in women than it was in men. He later reported that 60 percent of the 1400 antidepressant-treated patients had sexual dysfunction. According to personal interviews, up to 80 percent of patients affirmed that they had a disorder.
Anita Clayton has written a methodologically excellent paper. Anita Clayton reports that 40% of patients who have received SSRs are suffering from severe sexual dysfunction. dysfunction. She describes, gender differences. Tricyclic antidepressants, such as amitriptyline, are more difficult for men to rouse than SSRIs. SSRIs are more likely to induce anorgasmia among women and delay ejaculation for men. Masturbation is also subject to these limitations.
Jansen-Cilag created Dapotin, an SSRI which has no indications as an antidepressant but can be used to treat premature ejaculation. Dapotin, also known as the SSRI for urology, is Dapotin. It is “supposed to” cause sexual excitability. Experts disagree on whether premature ejaculation really is a “disease” or whether drug treatment is the best option. Method of treatment.
Some pharmaceutical companies saw a market opportunity for non-SSRI antidepressants due to the sexual problem. They sent their representatives out to promote Bupropin. Bupropion, Mirtazapine, Agomelatin, or any other drug, is not something that informed people should be impressed with. Experts agree that sexual dysfunction is the leading reason patients stop taking their antidepressants. This is true for all antidepressants.
SSRIs are not the only drugs that can cause sexual impairment. All agents that affect 5-HT2C or 5-HT3 receptors can impair the sexual function. However, drugs that also act on ACh receptors and dopamine D2 receptors or block nitrogen synthesis can cause impairment. Since long, the pharmaceutical industry has known that antidepressants can cause impotence. Multiple studies have shown that Viagra can reverse the effects of SSRIs, phenelzine or fluvoxamine on impotence. These reports were published more than 20 years ago. However, your doctor may not have known until very recently that antidepressants could affect sexuality .
What does this leave us with? To recommend a specific substance, you must believe in the power of selectivity of molecules. As biochemists repeatedly demonstrated, not all antidepressant drugs can be as selective as pharmaceutical companies claim. They can be used at nearly all receptors at once.
Chemical drugs always cause side effects but Eroxel being completely natural is totally effective, without causing you any extra harm: Eroxel test